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Romac (Roxithromycin)
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Roxithromycin

Romac is a semi-synthetic 14-membered-ring macrolide antibiotic in which the erythronolide A lactone ring has been altered to prevent inactivation in the gastric milieu. The in-vitro activity of Romac is well documented and similar to that of other macrolide antibiotics. Romac is active against Gram-positive and Gram-negative cocci, Gram- positive bacilli and some Gram-negative bacilli, but has no significant effect on the pre- dominant faecal flora. It also displays good activity against atypical pathogens, such as Mycobacterium avium complex, Helicobacter pylori and Borrelia spp. It penetrates and accumulates within cells, such as macrophages and polymorphonuclear neutrophils (PMNs), where it is distributed between the cytosol and cellular granules. Once inside the cells, it is active against intracellular pathogens, such as Legionella, Chlamydia, Mycobacterium, Rickettsia and Borrelia spp. Like other macrolides, Romac displays a significant post- antibiotic effect which is dependent on the pathogens under study, the concentration of Romac and the duration of exposure. In vivo, Romac is as effective or more effective than other macrolides in a wide range of infections.

Other names for this medication:
Roxithromycin, Rulide, Biaxsig, Ixor, Makrodex, Nirox, Overal, Remora, Renicin, Romac, Roxithromycine, Rulid,

Similar Products:
Ogasto, Gastec, Helicid, Olit, Duloxetine, Epivir, Esita, Sanprima, Aldactone, Neurexal, Citox, Biclar, Moxypen, Cilift, Recamicina, Largopen, Makrodex, Omax, Ilsatec, Donaren, Tipuric, Lipophage, Macladin, Pantocalm, Amoxin, Citalec, Dresplan, Pripsen, Prograf,

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To unravel the role of PMNs during the early phase of infection, we depleted mice of PMNs by using an anti-Ly6G antibody, after which we could see more-efficient initial colonization by the wild-type strain as well as by yopH, yopE, and yopK mutants on day 1 postinfection. In some respects, Sweden is becoming similar to other Organisation for Economic Co-operation and Development countries. They have to be preoperatively identified in order to modify the procedure of an eventual mediastinal surgery. The above inconsistencies between observations in pressure-flow kinetics and ordinary passive diffusion indicate that either the current model should be adjusted or a new model should be proposed. Clinical application of cultured oral epithelium for palatal wounds after palatoplasty: a preliminary report.

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